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Alcohol consumption, metabolic syndrome and liver cancer
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[1]Donato F, GelattiU, Limina RM, et al.Southern Europe as an ex-ample of interaction between various environmental factors:a system-atic reiew of the epidemiologic evidence[J].Oncogene2006, 25 (27) ∶3756-3770. [2]Marrero JA, Fontana RJ, Fu S, et al.Alcohol, tobacco and obesity are synergistic risk factors for hepatocellular carcinoma[J].Hepatol, 2005, 42 (2) 218-224. [3]丁建华等.醇醛脱氢酶基因多态和饮酒习惯与肝癌易感性[J].中华肿瘤防治杂志, 2007, 14 (7) :500-504. [4]Yoshiyuki Y, Horie Y, Kajihara M, et al.Hepatocellular carcinoma in heavy drinkers with negative markers for viral hepatitis[J].Hepa-tol.2004, 28 (4) ∶177-183. [5]Paradis V, Zalinskis, Chelbi E, et al.Hepatocellular Carcinomas in Patients With Metabolic Syndrome Often Develop Without Significant Liver Fibrosis:A Pathological Analysis[J].Hepatology, 2009, 49 (3) ∶851-859. [6]El-serag, HB.Rudolph KL.Hepatocellular Carcinoma:Epidemi-ology and Molecular Carcinogenesis[J].Gastrenterology, 2007, 132 (7) ∶2557-2576. [7]季尚玮, 王江滨.2型糖尿病对原发性肝癌发病的危险性分析[J].肝脏, 2007, 12 (3) :168-170. [8]Lemke LB, Rogers AB, Nambiar PR, et al.Obesity and non-insu-lin-dependent diabetes mellitus in Swiss-Webster mice associated with late-onset hepatocellular carcinoma[J].Endocrinol, 2008, 199 (1) ∶21-32. [9]Chen CL, Yang HI, Yang WS, et al.Metabolic Factors and Risk of Hepatocellular Carcinoma by Chronic Hepatitis B/C Infection:AFol-low-up Study in Taiwan[J].Gastroenterology, 2008, 135 (1) ∶111-121. -
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