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A report of severe hepatitis with wilson desease and hepatitis B
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摘要: <正>肝豆状核变性是常染色体隐性遗传性疾病,其临床表现与病毒性肝炎相似,很容易引起误诊,当病毒标志物阳性时,很容易忽视对血清铜蓝蛋白和血清铜的检测,而误诊为肝炎。我科近期成功抢救一例肝豆状核变性合并乙型肝炎致重型肝炎的患者,现报道如下。
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Key words:
- hepatolenticular degeneration /
- hepatitis B /
- Unithiol
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[1]马仕坤, 汪荣泉.肝豆状核变性误诊为药物性肝损害1例[J].第三军医大学学报, 2009, 31 (17) ∶1675-1684. [2]Okada T, Morise T, Takeda Y, et al.A new variant deletion of a copper-transporting P-type ATP ase gene found in patients with Wilson's disease presenting with fulminant hepatic failure[J].J Gastroenterol, 2000, 35 (4) ∶278-283. [3]Sallie R, Chiyende J, Tan KC, et al.Fulminant hepatic failure resulting from coexistent Wison'disease and hepatitis E[J].Gut, 1994, 35 (6) ∶849-853. [4]王晓平.巯甲丙脯酸与二巯基丙磺酸钠治疗肝豆状核变性[J].中华肝脏病杂志, 2002, 10 (10) ∶389. [5]王晓红.肝豆状核变性疾病治疗规范的变革-锌剂成为首选治疗[J].国外医学医学地理分册, 2006, 12 (27) ∶168-170. [6]Ala A, Walker AP, Ashkan K, et al.Wilson’s disease[J].Lancet, 2007, 369 (9559) ∶397-408. [7]Linn FH, Houwen RH, van Hattum J, et al.Long-term exclusive zinc monotherapy in symptomatic Wilson disease:experience in17patients[J].Hepatology, 2009, 50 (5) ∶1442-1452. -
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