|
[1] EKANAYAKA SA, MCCLELLAN SA, PENG XD, et al. HMGB1antagonist, box A, reduces TLR4, RAGE, and inflammatorycytokines in the cornea of P. aeruginosa-infected mice[J]. JOcul Pharmacol Ther, 2018, 34 (10) :1-9.
|
|
[2] LU B, NAKAMURA T, INOUYE K, et al. Novel role of PKR ininflammasome activation and HMGB1 release[J]. Nature, 2012, 488 (7413) :670-674.
|
|
[3] YANG H, WANG H, JU Z, et al. MD-2 is required for disul-fide HMGB1-dependent TLR4 signaling[J]. J Exp Med, 2015, 212 (1) :5-14.
|
|
[4] YANG H, LUNDBCK P, OTTOSSON L, et al. Redox modifi-cation of cysteine residues regulates the cytokine activity ofhigh mobility group box-1 (HMGB1) [J]. Mol Med, 2016, 18 (1) :250-259.
|
|
[5] TANG D, KANG R, ZEH HJ, et al. High-mobility group box1, oxidative stress, and disease[J]. Antioxid Redox Signal, 2011, 14 (7) :1315-1335.
|
|
[6] TANG D, KANG R, LIVESEY KM, et al. Endogenous HMGB1regulates autophagy[J]. J Cell Biol, 2010, 190 (5) :881-892.
|
|
[7] HAO N, BUDNIK BA, GUNAWARDENA J, et al. Tunable sig-nal processing through modular control of transcription factortranslocation[J]. Science, 2013, 339 (6118) :460-464.
|
|
[8] ZHOU H, JIN C, CUI L, et al. HMGB1 contributes to the irra-diation-induced endothelial barrier injury through receptor foradvanced glycation endproducts (RAGE) [J]. J Cell Physiol, 2018, 233 (9) :6714-6721.
|
|
[9] SUNG PH, LEE FY, LIN LC, et al. Melatonin attenuated braindeath tissue extract-induced cardiac damage by suppressingDAMP signaling[J]. Oncotarget, 2018, 9 (3) :3531-3548.
|
|
[10] MIHM S. Danger-associated molecular patterns (DAMPs) :Molecular triggers for sterile inflammation in the liver[J]. Int JMol Sci, 2018, 19 (10) :1-12.
|
|
[11] LV WL, ARNESANO F, CARLONI P, et al. Effect of in vivopost-translational modifications of the HMGB1 protein uponbinding to platinated DNA:A molecular simulation study[J].Nucleic Acids Res, 2018, 46 (22) :11687-11697.
|
|
[12] VENEREAU E, DE LF, MEZZAPELLE R, et al. HMGB1 as bio-marker and drug target[J]. Pharmacol Res, 2016, 111:534-544.
|
|
[13] LIU A, DIRSCH O, FANG H, et al. HMGB1 translocation andexpression is caused by warm ischemia reperfusion injury, butnot by partial hepatectomy in rats[J]. Exp Mol Pathol, 2011, 91 (2) :502-508.
|
|
[14] LIU A, DIRSCH O, FANG H, et al. HMGB1 in ischemic andnon-ischemic liver after selective warm ischemia/reperfusionin rat[J]. Histochem Cell Biol, 2011, 135 (5) :443-452.
|
|
[15] CANNISTRM, RUGGIERO M, ZULLO A, et al. Hepatic is-chemia reperfusion injury:A systematic review of literature andthe role of current drugs and biomarkers[J]. Int J Surg, 2016, 33 (Suppl 1) :s57-s70.
|
|
[16] CUI Z, LI S, LIU Z, et al. Interferon regulatory factor 1 acti-vates autophagy to aggravate hepatic ischemia-reperfusioninjury by increasing high mobility group box 1 release[J]. CellPhysiol Biochem, 2018, 48 (1) :328-338.
|
|
[17] ZHU JR, LU HD, GUO CH, et al. Berberine attenuates ische-mia-reperfusion injury through inhibiting HMGB1 release andNF-κB nuclear translocation[J]. Acta Pharmacol Sin, 2018, 39 (11) :1706-1715.
|
|
[18] WU K, ZHANG H, FU Y, et al. TLR4/MyD88 signaling deter-mines the metastatic potential of breast cancer cells[J]. MolMed Rep, 2018, 18 (3) :3411-3420.
|
|
[19] CHEN BH, LI WM. Effect of ulinastatin combined with ische-mic preconditioning on hepatic ischemia-reperfusion injury inrats[J]. J Clin Hepatol, 2018, 34 (2) :369-372. (in Chi-nese) 陈宝鹤, 李文美.乌司他丁联合缺血预处理对大鼠肝缺血再灌注损伤的影响[J].临床肝胆病杂志, 2018, 34 (2) :369-372.
|
|
[20] SHAKER ME, TRAWICK BN, MEHAL WZ. The novel TLR9 an-tagonist COV08-0064 protects from ischemia/reperfusion in-jury in non-steatotic and steatotic mice livers[J]. BiochemPharmacol, 2016, 112:90-101.
|
|
[21] HUANG H, NACE GW, MCDONALD KA, et al. Hepatocyte-specific high-mobility group box 1 deletion worsens the injuryin liver ischemia/reperfusion:A role for intracellular high-mobility group box 1 in cellular protection[J]. Hepatology, 2014, 59 (5) :1984-1997.
|
|
[22] TSAROUCHA AK, VALSAMI G, KOSTOMITSOPOULOS N, etal. Silibinin effect on Fas/FasL, HMGB1, and CD45 expres-sions in a rat model subjected to liver ischemia-reperfusioninjury[J]. J Invest Surg, 2017:1-12.
|
|
[23] LI X, WU Y, ZHANG W, et al. Pre-conditioning with tanshi-none IIA attenuates the ischemia/reperfusion injury caused byliver grafts via regulation of HMGB1 in rat Kupffer cells[J]. Bi-omed Pharmacother, 2017, 89:1392-1400.
|
|
[24] TONG Y, TANG Z, YANG T, et al. Ulinastatin preconditioningattenuates inflammatory reaction of hepatic ischemia reperfu-sion injury in rats via high mobility group box 1 (HMGB1) inhi-bition[J]. Int J Med Sci, 2014, 11 (4) :337-343.
|
|
[25] YANG H, OCHANI M, LI J, et al. Reversing established sep-sis with antagonists of endogenous high-mobility group box 1[J]. Proc Natl Acad Sci U S A, 2004, 101 (1) :296-301.
|
|
[26] YAMAMOTO T, ONO T, ITO T, et al. Hemoperfusion with ahigh-mobility group box 1 adsorption column can prevent theoccurrence of hepatic ischemia-reperfusion injury in rats[J].Crit Care Med, 2010, 38 (3) :879-888.
|
DownLoad: