[1]KIM YR, LEE YH, CHOI KH, et al.Imaging findings of IgG4-related hepatopathy:A rare case presenting as a hepatic mass[J].Clin Imaging, 2018, 51:248-251.
|
[2]KAWA S, OTA M, YOSHIZAWA K, et al.HLA DRB10405-DQB10401 haplotype is associated with autoimmune pancreatitis in the Japanese population[J].Gastroenterology, 2002, 122 (5) :1264-1269.
|
[3]PARK DH, KIM MH, OH HB, et al.Substitution of aspartic acid at position 57 of the DQbeta1 affects relapse of autoimmune pancreatitis[J].Gastroenterology, 2008, 134 (2) :440-446.
|
[4]CULVER EL, CHAPMAN RW.IgG4-related hepatobiliary disease:An overview[J].Nat Rev Gastroenterol Hepatol, 2016, 13 (10) :601-612.
|
[5]CHANG MC, CHANG YT, TIEN YW, et al.T-cell regulatory gene CTLA-4 polymorphism/haplotype association with autoimmune pancreatitis[J].Clin Chem, 2007, 53 (9) :1700-1705.
|
[6]KOCHI Y, YAMADA R, SUZUKI A, et al.A functional variant in FCRL3, encoding Fc receptor-like 3, is associated with rheumatoid arthritis and several autoimmunities[J].Nat Genet, 2005, 37 (5) :478-485.
|
[7]UMEMURA T, OTA M, HAMANO H, et al.Genetic association of Fc receptor-like 3 polymorphisms with autoimmune pancreatitis in Japanese patients[J].Gut, 2006, 55 (9) :1367-1368.
|
[8]OGUCHI T, OTA M, ITO T, et al.Investigation of susceptibility genes triggering lachrymal/salivary gland lesion complications in Japanese patients with type 1 autoimmune pancreatitis[J].PLo S One, 2015, 10 (5) :e127078.
|
[9]OTA M, UMEMURA T, KAWA S.Immunogenetics of IgG4-Related AIP[J].Curr Top Microbiol Immunol, 2017, 401:35-44.
|
[10]OGUCHI T, OTA M, ITO T, et al.Correction:Investigation of susceptibility genes triggering lachrymal/salivary gland lesion complications in Japanese patients with type 1 autoimmune pancreatitis[J].PLo S One, 2016, 11 (1) :e146738.
|
[11]GUARNERI F, GUARNERI C, BENVENGA S.Helicobacter pylori and autoimmune pancreatitis:Role of carbonic anhydrase via molecular mimicry?[J].J Cell Mol Med, 2005, 9 (3) :741-744.
|
[12]FRULLONI L, LUNARDI C, SIMONE R, et al.Identification of a novel antibody associated with autoimmune pancreatitis[J].N Engl J Med, 2009, 361 (22) :2135-2142.
|
[13]FELIX K, HAUCK O, SCHNOLZER M, et al.Identification of novel serum autoantibodies for differential diagnosis of autoimmune pancreatitis and pancreatic ductal adenocarcinoma[J].Pancreas, 2016, 45 (9) :1309-1319.
|
[14]CULVER EL, SMIT WL, EVANS C, et al.No evidence to support a role for helicobacter pylori infection and plasminogen binding protein in autoimmune pancreatitis and IgG4-related disease in a UK cohort[J].Pancreatology, 2017, 17 (3) :395-402.
|
[15]WATANABE T, YAMASHITA K, FUJIKAWA S, et al.Involvement of activation of toll-like receptors and nucleotide-binding oligomerization domain-like receptors in enhanced IgG4responses in autoimmune pancreatitis[J].Arthritis Rheum, 2012, 64 (3) :914-924.
|
[16]MOISINI I, DAVIDSON A.BAFF:A local and systemic target in autoimmune diseases[J].Clin Exp Immunol, 2009, 158 (2) :155-163.
|
[17]NARADIKIAN MS, PERATE AR, CANCRO MP.BAFF receptors and ligands create independent homeostatic niches for Bcell subsets[J].Curr Opin Immunol, 2015, 34:126-129.
|
[18]WATANABE T, YAMASHITA K, SAKURAI T, et al.Toll-like receptor activation in basophils contributes to the development of IgG4-related disease[J].J Gastroenterol, 2013, 48 (2) :247-253.
|
[19]AKITAKE R, WATANABE T, ZAIMA C, et al.Possible involvement of T helper type 2 responses to Toll-like receptor ligands in IgG4-related sclerosing disease[J].Gut, 2010, 59 (4) :542-545.
|
[20]KIYAMA K, KAWABATA D, HOSONO Y, et al.Serum BAFFand APRIL levels in patients with IgG4-related disease and their clinical significance[J].Arthritis Res Ther, 2012, 14 (2) :R86.
|
[21]FURUKAWA S, MORIYAMA M, TANAKA A, et al.Preferential M2 macrophages contribute to fibrosis in IgG4-related dacryoadenitis and sialoadenitis, so-called Mikulicz's disease[J].Clin Immunol, 2015, 156 (1) :9-18.
|
[22]ZEN Y, FUJII T, HARADA K, et al.Th2 and regulatory immune reactions are increased in immunoglobin G4-related sclerosing pancreatitis and cholangitis[J].Hepatology, 2007, 45 (6) :1538-1546.
|
[23]UMEHARA H, NAKAJIMA A, NAKAMURA T, et al.IgG4-related disease and its pathogenesis-cross-talk between innate and acquired immunity[J].Int Immunol, 2014, 26 (11) :585-595.
|
[24]HEERINGA JJ, KARIM AF, van LAAR J, et al.Expansion of blood IgG4 (+) B, TH2, and regulatory T cells in patients with IgG4-related disease[J].J Allergy Clin Immunol, 2018, 141 (5) :1831-1843.
|
[25]LI YH, XIANG XX.Research advances in immunoglobulin G4-related sclerosing cholangitis[J].J Clin Hepatol, 2017, 33 (11) :2239-2242. (in Chinese) 李雨涵, 向晓星.IgG4相关性硬化性胆管炎的研究现状[J].临床肝胆病杂志, 2017, 33 (11) :2239-2242.
|
[26]DELLA-TORRE E, LANZILLOTTA M, DOGLIONI C.Immunology of IgG4-related disease[J].Clin Exp Immunol, 2015, 181 (2) :191-206.
|
[27]NAKASHIMA H, MIYAKE K, MORIYAMA M, et al.An amplification of IL-10 and TGF-beta in patients with IgG4-related tubulointerstitial nephritis[J].Clin Nephrol, 2010, 73 (5) :385-391.
|
[28]OHTA N, KURAKAMI K, ISHIDA A, et al.Roles of TGF-beta and periostin in fibrosclerosis in patients with IgG4-related diseases[J].Acta Otolaryngol, 2013, 133 (12) :1322-1327.
|
[29]CONWAY SJ, DOETSCHMAN T, AZHAR M.The inter-relationship of periostin, TGF beta, and BMP in heart valve development and valvular heart diseases[J].Scientific World J, 2011, 11:1509-1524.
|
[30]STONE JH, ZEN Y, DESHPANDE V.IgG4-related disease[J].N Engl J Med, 2012, 366 (6) :539-551.
|
[31]UCHIDA K, KUSUDA T, KOYABU M, et al.Regulatory T cells in type 1 autoimmune pancreatitis[J].Int J Rheumatol, 2012, 2012:795026.
|
[32]KUSUDA T, UCHIDA K, MIYOSHI H, et al.Involvement of inducible costimulator-and interleukin 10-positive regulatory Tcells in the development of IgG4-related autoimmune pancreatitis[J].Pancreas, 2011, 40 (7) :1120-1130.
|
[33]MAEHARA T, MATTOO H, MAHAJAN VS, et al.The expansion in lymphoid organs of IL-4 (+) BATF (+) T follicular helper cells is linked to IgG4 class switching in vivo[J].Life Sci Alliance, 2018, 1 (1) :e201800050.
|
[34]GASCAN H, GAUCHAT JF, RONCAROLO MG, et al.Human B cell clones can be induced to proliferate and to switch to IgEand IgG4 synthesis by interleukin 4 and a signal provided by activated CD4+T cell clones[J].J Exp Med, 1991, 173 (3) :747-750.
|
[35]WALLACE ZS, MATTOO H, CARRUTHERS M, et al.Plasmablasts as a biomarker for IgG4-related disease, independent of serum IgG4 concentrations[J].Ann Rheum Dis, 2015, 74 (1) :190-195.
|
[36]AKIYAMA M, YASUOKAH, YAMAOKA K, et al.Enhanced IgG4 production by follicular helper 2 T cells and the involvement of follicular helper 1 T cells in the pathogenesis of IgG4-related disease[J].Arthritis Res Ther, 2016, 18:167.
|
[37]LIN W, ZHANG P, CHEN H, et al.Circulating plasmablasts/plasma cells:A potential biomarker for IgG4-related disease[J].Arthritis Res Ther, 2017, 19 (1) :25.
|
[38]FOX RI, FOX CM.IgG4 levels and plasmablasts as a marker for IgG4-related disease (IgG4-RD) [J].Ann Rheum Dis, 2015, 74 (1) :1-3.
|
[39]MATTOO H, MAHAJAN VS, DELLA-TORRE E, et al.De novo oligoclonal expansions of circulating plasmablasts in active and relapsing IgG4-related disease[J].J Allergy Clin Immunol, 2014, 134 (3) :679-687.
|
[40]MAJUMDER S, MOHAPATRA S, LENNON R J, et al.Rituximab maintenance therapy reduces rate of relapse of pancreaticobiliary immunoglobulin G4-related disease[J].Clin Gastroenterol Hepatol, 2018, 16 (12) :1947-1953.
|
[41]DELLA-TORRE E, FEENEY E, DESHPANDE V, et al.B-cell depletion attenuates serological biomarkers of fibrosis and myofibroblast activation in IgG4-related disease[J].Ann Rheum Dis, 2015, 74 (12) :2236-2243.
|
[42]XIAO X, LIAN M, ZHANG W, et al.The Immunologic paradoxes of IgG4-related disease[J].Clin Rev Allergy Immunol, 2018, 54 (2) :344-351.
|
[43]MASAKI Y, DONG L, KUROSE N, et al.Proposal for a new clinical entity, IgG4-positive multiorgan lymphoproliferative syndrome:Analysis of 64 cases of IgG4-related disorders[J].Ann Rheum Dis, 2009, 68 (8) :1310-1315.
|
[44]TAKEUCHI M, SATO Y, OHNO K, et al.T helper 2 and regulatory T-cell cytokine production by mast cells:A key factor in the pathogenesis of IgG4-related disease[J].Mod Pathol, 2014, 27 (8) :1126-1136.
|
[45]CULVER EL, SADLER R, BATEMAN AC, et al.Increases in IgE, eosinophils, and mast cells can be used in diagnosis and to predict relapse of IgG4-related disease[J].Clin Gastroenterol Hepatol, 2017, 15 (9) :1444-1452.
|
[46]WALLACE ZS, MATTOO H, MAHAJAN VS, et al.Predictors of disease relapse in IgG4-related disease following rituximab[J].Rheumatology (Oxford) , 2016, 55 (6) :1000-1008.
|
[47]JACOBS JF, van der MOLEN RG, KEREN DF.Relatively restricted migration of polyclonal IgG4 may mimic a monoclonal gammopathy in IgG4-related disease[J].Am J Clin Pathol, 2014, 142 (1) :76-81.
|
[48]LIGHAAM LC, RISPENS T.The immunobiology of immunoglobulin G4[J].Semin Liver Dis, 2016, 36 (3) :200-215.
|
[49]TAKEUCHI A, SAITO T.CD4 CTL, a cytotoxic subset of CD4 (+) T Cells, their differentiation and function[J].Front Immunol, 2017, 8:194.
|
[50]LAIDLAW BJ, LU Y, AMEZQUITA RA, et al.Interleukin-10 from CD4 (+) follicular regulatory T cells promotes the germinal center response[J].Sci Immunol, 2017, 2 (16) :eaan4767.
|
[51] MATTOO H, MAHAJAN VS, DELLA-TORRE E, et al.De novo oligoclonal expansions of circulating plasmablasts in active and relapsing IgG4-related disease[J].J Allergy Clin Immunol, 2014, 134 (3) :679-687.
|
[52]MATTOO H, MAHAJAN VS, MAEHARA T, et al.Clonal expansion of CD4 (+) cytotoxic T lymphocytes in patients with IgG4-related disease[J].J Allergy Clin Immunol, 2016, 138 (3) :825-838.
|
[53]MAEHARA T, MATTOO H, OHTA M, et al.Lesional CD4+IFN-gamma+cytotoxic T lymphocytes in IgG4-related dacryoadenitis and sialoadenitis[J].Ann Rheum Dis, 2017, 76 (2) :377-385.
|
[54]MAEHARA T, MORIYAMA M, NAKAMURA S.Pathogenesis of IgG4-related disease:Acritical review[J].Odontology, 2018.
|
[55]KAMEKURA R, TAKAHASHI H, ICHIMIYA S.New insights into IgG4-related disease:emerging new CD4+T-cell subsets[J].Curr Opin Rheumatol, 2019, 31 (1) :9-15.
|
[56]KUBO S, NAKAYAMADA S, ZHAO J, et al.Correlation of Tfollicular helper cells and plasmablasts with the development of organ involvement in patients with IgG4-related disease[J].Rheumatology (Oxford) , 2018, 57 (3) :514-524.
|
[57]CHEN Y, LIN W, YANG H, et al.Aberrant expansion and function of follicular helper T cell subsets in IgG4-related disease[J].Arthritis Rheumatol, 2018, 70 (11) :1853-1865.
|
[58]KAMEKURA R, TAKANO K, YAMAMOTO M, et al.Cutting edge:A critical role of lesional T follicular helper cells in the pathogenesis of IgG4-related disease[J].J Immunol, 2017, 199 (8) :2624-2629.
|
[59]KAMEKURA R, SHIGEHARA K, MIYAJIMA S, et al.Alteration of circulating type 2 follicular helper T cells and regulatory Bcells underlies the comorbid association of allergic rhinitis with bronchial asthma[J].Clin Immunol, 2015, 158 (2) :204-211.
|
[60]AKIYAMA M, SUZUKI K, YAMAOKA K, et al.Number of circulating follicular helper 2 T cells correlates with IgG4 and interleukin-4 levels and plasmablast numbers in IgG4-related disease[J].Arthritis Rheumatol, 2015, 67 (9) :2476-2481.
|
[61]GRAHAM RP, SMYRK TC, CHARI ST, et al.Isolated IgG4-related sclerosing cholangitis:A report of 9 cases[J].Hum Pathol, 2014, 45 (8) :1722-1729.
|
[62]ZEN Y, LIBERAL R, NAKANUMA Y, et al.Possible involvement of CCL1-CCR8 interaction in lymphocytic recruitment in IgG4-related sclerosing cholangitis[J].J Hepatol, 2013, 59 (5) :1059-1064.
|
[63]SELEZNIK GM, REDING T, ROMRIG F, et al.Lymphotoxin beta receptor signaling promotes development of autoimmune pancreatitis[J].Gastroenterology, 2012, 143 (5) :1361-1374.
|